In this section I will explore the arguments of pro-vaccinator's against actual medical evidence. These pro-vaccine arguments come from BCCDC Centre for Disease Control,Provincial Health Services Authority, Vancouver - www.bccdc.org
Weren't the diseases disappearing long before vaccines became available?
No. A common argument of those opposed to immunization is that vaccine-preventable diseases were declining in frequency in Canada and other developed countries long before vaccines became available. This is not true. Until vaccines became available, there was no significant change in the numbers of cases of diphtheria, tetanus, pertussis, polio, measles, mumps, rubella, Hib or hepatitis B.
What was changing before vaccines became available was the death rate from some of these diseases. Improvements in social and economic conditions led to declining death rates for many common diseases. Children who are healthy and well-nourished are much less likely to die from measles or pertussis than malnourished children.
VAN UK'S Comment
Both disease rates and death rates from diseases were decreasing prior to vaccination, and in some cases, figures showed an increase in disease after vaccination was introduced, for instance, in this US graph for Diphtheria disease and death rates:
If you notice, when the vaccine was introduced in the 1920's, the disease and death rates spiked on the graph, before reducing again, suggesting a relation to the vaccine.
All diseases have become milder throughout history, and mortality rates have reduced and it's nothing to do with vaccination. For instance, in the 14th century the Bubonic Plague ('the black death') killed 25 million Europeans in just 5 years. Now that's an epidemic! Now people no longer die of it, and there was no vaccine to credit it's decline.
Typhus also disappeared without any vaccine, as did Scarlet Fever.
The Archives of Pediatrics, in 1951, stated:
'The decline in diphtheria, whooping cough and typhoid fever began fully fifty years prior to the inception of artificial immunization and followed an almost even grade before and after the adoption of these control measures. In the case of scarlet fever, mumps, measles and rheumatic fever there has been no specific innovation in control measures, yet these also have followed the same general pattern in incidence decline. Claims about the historical life-saving impact of immunization programs appear to be assumptive and not factual.'
McCormick W.J., Vitamin C in the Prophylaxis and Therapy of Infectious Diseases; Archives of Pediatrics, Vol. 68, No. 1, January 1951.
The above graph shows the decline of Scarlet Fever, starting in the 1880's, which occured without any vaccination.
Yet vaccination is taking the credit for the decline of many diseases, when evidence suggests that the natural pattern for the diseases would be to reduce in numbers and severity anyway.
The second paragraph of the pro-vaccinator's argument is true. Deaths were declining, and the reason was better sanitation and nutrition.
One of the main arguments that the medical profession use to get parents to consent to vaccination is the fear that the child might DIE of a vaccine-preventable disease, and they say that vaccination is to prevent deaths, save lives etc. For instance, Professor Ian Frazer, inventor of the HPV vaccine, said
"Everybody has the right to say no, that is their right but they also have the right to get cervical cancer and they also have the right to die, it goes with the territory.'
He is selling his vaccine to parents by saying their daughters will die if they don't have it. No parent would accept a vaccine for an illness they thought their child would easily recover from. So in fact, the declining death rates are of more importance than the declining disease rates.
Do vaccines really work?
Yes! All vaccine-preventable diseases have declined significantly in countries with successful immunization programs. Wherever vaccination rates are high, disease rates are low.
VAN UK'S Comment
This is a very vague statement with no sources to back it up.
Here are dozens of referenced medical studies which show that diseases are occuring in the vaccinated:
Manghani DK, et al. Pleomorphism of fine structure of rabies virus in human and experimental brain. J Neurol Sci. 1986 Sep;75(2):181-93. PMID: 3760910; UI: 87010723.
Identification of the Negri bodies in the brain of an 8-year-old boy who died 8 days after a paralytic illness and 20 days after a dog bite, and who had received 9 injections of Semple's anti-rabies vaccine, provided evidence that he died of acute rabies encephalitis and not of post-vaccinal allergic encephalomyelitis.
"Reemergence of invasive haemophilus influenzae type b disease in a well-vaccinated population on remote Alaska" (Journal of Infectious Diseases, vol. 179, no. 1, January 1999, pp. 101-106.
"High incidence of breakthrough varicella observed in healthy Japanese children immunized with live attenuated varicella vaccine (Oka strain)," Acta Paediatrica Japonica, vol. 39, no. 6, December 1997, pp. 663-8: the rate of varicella [chicken pox] occurrence among vaccinees was found to be much higher than rates reported previously by other authors. "Varicella vaccine seems to be effective in modifying the symptoms of varicella, but not potent enough in protecting from VZV infection."
The characteristics of poliovirus strains circulating in Ukraine in 1982-1994" (Mikrobiol[ogie] Z. vol. 60, no. 2, March-April 1998, pp. 44-49 [article in Russian]): "The long-term use of the live poliomyelitis vaccine has not stopped circulation of virulent polioviruses."
Rev. Soc. Bras. Med. Trop., vol. 28, no. 4, Oct-Dec 1995, pp. 339-43 "Clinical and epidemiological findings during a measles outbreak occurring in a population with a high vaccination coverage" : "The history of previous vaccination [in very early childhood] did not diminish thenumber of complications of the cases studied. The results of this work show changes in age distribution of measles leading to sizeable outbreaks among teenagers and young adults."
Clin. Invest. Med., vol. 11, no. 4, August 1988, pp. 304-9: "Measles serodiagnosis during an outbreak in a vaccinated community" ( from a group of 30 measles-sufferers displaying IgM antibodies during the acute phase of illness, 17 had been vaccinated for measles. All 17 experienced measles again, showing IgM antibodies indicating acute infection. "A history of prior vaccination is not always associated with immunity nor with the presence of specific antibodies."
Boulianne N, et al.(1991) [Major measles epidemic in the region of Quebec despite a 99% vaccine coverage]. Can J Public Health. 1991 May-Jun;82(3):189-90. French. PMID: 1884314; UI: 91356447.
The 1989 measles outbreak in the province of Quebec has been largely attributed to an incomplete vaccination coverage. In the Quebec City area (pop. 600,000) 1,363 confirmed cases of measles did occur. A case-control study conducted to evaluate risk factors for measles allowed us to estimate vaccination coverage. It was measured in classes where cases did occur during the outbreak. This population included 8,931 students aged 5 to 19 years old. The 563 cases and a random sample of two controls per case selected in the case's class were kept for analysis. The vaccination coverage among cases was at least 84.5%. Vaccination coverage for the total population was 99.0%. Incomplete vaccination coverage is not a valid explanation for the Quebec City measles outbreak.
Edmonson MB, et al.(1990) Mild measles and secondary vaccine failure during a sustained outbreak in a highly vaccinated population. JAMA. 1990 May 9;263(18):2467-71. PMID: 2278542; UI: 90230400.
A prolonged school-based outbreak of measles provided an opportunity to study "vaccine-modified" mild measles and secondary vaccine failure. Thirty-six (97%) of 37 unvaccinated patients had rash illnesses that met the Centers for Disease Control clinical case definition of measles, but 29 (15%) of 198 vaccinated patients did not, primarily because of low-grade or absent fever. Of 122 patients with seroconfirmed measles, 10 patients (all previously vaccinated) had no detectable measles-specific IgM and significantly milder illness than either vaccinated or unvaccinated patients with IgM-positive serum. Of 108 vaccinated patients with seroconfirmed measles, 17 patients (16%) had IgM-negative serology or rash illnesses that failed to meet the clinical case definition; their mean age (13 years), age at the time of vaccination, and time since vaccination did not differ from those of other vaccinated patients. The occurrence of secondary vaccine failure and vaccine-modified measles does not appear to be a major impediment to measles control in the United States but may lead to underreporting of measles cases and result in overestimation of vaccine efficacy in highly vaccinated populations. http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2278542&form=6&db=m&Dopt=b
Journal of Infectious Diseases, vol. 179, April 1999; 915-923. "Temporal trends in the population structure of bordetella pertussis during 1949-1996 in a highly vaccinated population "Despite the introduction of large-scale pertussis vaccination in 1953 and high vaccination coverage, pertussis is still an endemic disease in The Netherlands, with epidemic outbreaks occurring every 3-5 years." One factor that might contribute to this is the ability of pertussis strains to adapt to vaccine-induced immunity, causing new strains of pertussis to re-emerge in this well-vaccinated population.
"Vaccination against whooping-cough. Efficacy versus risks" (The Lancet, vol. 1, January 29, 1977, pp. 234-7): Calculations based on the mortality of whooping-cough before 1957 predict accurately the subsequent decline and the present low mortality
Incidence [is] unaffected either by small-scale vaccination beginning about 1948 or by nationwide vaccination beginning in 1957
No protection is demonstrable in infants."
Bentsi-Enchill AD, et al. Estimates of the effectiveness of a whole-cell pertussis vaccine from an outbreak in an immunized population. Vaccine. 1997 Feb;15(3):301-6. PMID: 9139490; UI: 97227584.
D. C. Christie, et al., "The 1993 Epidemic of Pertussis in Cincinnati: Resurgence of Disease in a Highly Immunized Population of Children," New England Journal of Medicine (July 7, 1994), pp. 16-20.
The Lancet Volume 353, Number 9150 30 January 1999 Risk of diphtheria among schoolchildren in the Russian Federation in relation to time since last vaccination
In 1993, the Russian Federation reported 15229 cases of diphtheria, a 25-fold increase over the 603 cases reported in 1989.1 The incidence rate among children 7-10 years of age (15·7 per 100000) was twice that of adults aged 18 years or over (7·9 per 100000).4 81% of the affected children aged 7-10 years had been vaccinated with at least a primary series of diphtheria toxoid, and most had received the first booster recommended to be given 12 months after completion of the primary series.
Need I go on? Medical journals have been jammed full of these failure reports in vaccinated populations since vaccines were invented, only in those old days, the vaccine simply would have been called 'spurious' if the recipient got the disease (i.e. that one was a dud. Don't worry, vaccination still works).
Why do some children still get measles after they've been vaccinated?
We know that one dose of measles vaccine is not 100% effective. We know that about 5-10% of children are not protected after a single dose of vaccine.
Whenever a vaccine is not 100% effective and most children have been vaccinated, more cases will occur in vaccinated than in unvaccinated children during outbreaks, only because there are more vaccinated than unvaccinated children.
VAN UK'S Comment
When vaccines were first introduced, we were told one shot was for life. The same was true for the MMR. Then they introduced a booster and now in some countries they have an additional shot at 12 years old.
My question is, if a child does not become immune from one dose of vaccine, why would he become immune from a second dose? If it didn't work the first time, there is no reason to assume it would the second time.
It is true that there are more vaccinated children than unvaccinated, and therefore more vaccinated children are getting diseases (it still does not look good for vaccination, since those children were supposed to be 'immunized').
According to Bailey GVJ, Narain R, Mayurnath S, Vallisliayee SRS, GuldJ. Tuberculosis prevention trial, Madras Inj J Med Res 72 (suppl)Jul 1980:1-74, there was the world's one and only double-blinded trial on BCG vaccination where one group was totally unvaccinated and one group had the BCG vaccine.
There were more cases of TB in the vaccine group! This suggests that it is not just that there are more vaccinated than unvaccinated, but that the vaccine may actually be causing TB.
The trial was abruptly stopped and such a trial on any vaccine has never been repeated because doctors say to leave a child without vaccines would be 'unethical'. I think the real reason is they would get more results like the BCG trial and they don't want to know that vaccines cause disease.
How can immunization, which is artificial, be as good as immunity resulting from natural infection?
Opponents ignore the fact that infection always has a much greater risk of causing harm than does immunization. There are two functions of the immune system: immediate response and long-term response. The immediate response kills infectious germs and promotes recovery from the infection. The long-term response maintains immunity so that the person will be protected against infection if exposed to the organisms in the future.
VAN UK'S Comment
There IS greater harm from a vaccine than the disease, because:
1. Not every unvaccinated person will get the disease, but EVERY vaccinated person is at risk of vaccine induced complications.
2. Vaccines don't just contain parts of a virus, they have toxic chemicals in them. For instance, the HPV vaccine has sodium borate in it which is ant powder and not meant for internal use. These chemicals can injure just on their own without even considering the vaccine virus.
3. Some of the diseases are mild have symptoms less severe than the vaccine. For instance, here are the symptoms for Chickenpox as written on the NHS's website, NHS Direct:
'Chickenpox is a mild, but highly infectious disease that most children catch at some point. It is most common to catch the disease between March and May. It takes 10-21 days for the symptoms to show after you have come into contact with the virus. This is called the 'incubation period'.
Chickenpox is most common in children who are between 2-8 years of age, although you can develop chickenpox at any age. You are infectious from about two days before the rash appears until roughly five days after. Therefore, you, or your child, should stay at home until all of the blisters have fully crusted over, and this usually happens 5-7 days after the first blister appears. After the last blister has burst and crusted over, you are no longer infectious.
Chickenpox spreads in tiny droplets of saliva and nasal mucus, by sneezes and coughs from an infected person. The virus is already in these droplets, which is why it spreads so fast.
Once you have had chickenpox, you will very rarely catch chickenpox for a second time. This is because your body develops immunity to the chickenpox virus, which stops you from becoming re-infected. '
And here is a manufacturer's data sheet for Varivax Chickenpox vaccine and the side-effects (Sanofi Pasteur MSD):
Common side effects include:
Pain, tenderness, soreness, erythema, swelling and pruritus at the injection site,
varicella-like rash, upper respiratory infection, irritability, rash and fever.
Rarely, serious side effects have been reported and include thrombocytopenia,
pneumonia, pulmonary congestion, encephalitis, anaphylaxis, cerebrovascular
accident, convulsions, Guillain-Barré syndrome, transverse myelitis, ataxia,
Stevens-Johnson syndrome, erythema multiforme and Henoch-Schönlein
purpura. The vaccine virus may rarely be transmitted to contacts of vaccinees
who develop a varicella-like rash.
Which one do you think sounds more serious?
Does immunity wear off over time?
The levels of antibody in the blood decline over time following both natural infection and vaccination. But even though antibodies disappear, immune memory persists. Most vaccines produce immune memory that lasts a very long time, if not for life. The presence of immune memory means that the immune system can respond very quickly, to either infection or a booster dose of vaccine.
VAN UK'S Comment
That is nonsense. Vaccine immunity is based on antibody count and nothing else. If the body had 'immune memory' to vaccination, it would then produce antibodies as a result. It is a documented fact that these wane weeks or months after vaccination, which is why adults are encouraged to have tetanus vaccine every 10 years because of declining antibodies. The 'immune memory' spoken about by scientists may in fact be nothing more than over-sensitization to vaccine agents.
Waning antibodies are also the reason why boosters are introduced, and why the UK government is now giving some babies two doses of MMR at once, because they say that it'll 'protect' the babies from those babies who have not had MMR.
Not only does vaccine immunity wane, but if a mother has booster vaccines during pregnancy, she can put her child at risk of disabilities. In the paper, Adverse Outcomes Associated with Postpartum Rubella or MMR Vaccine, F. Edward Yazbak, MD, FAAP and Kathy L. Lang-Radosh, MS, it states:
'We identified 60 rubella-susceptible mothers who were revaccinated in the postpartum period with either the measles-mumps-rubella (MMR) or the monovalent rubella vaccine and whose children later received MMR vaccine. Forty-five of these women have children diagnosed with autistic spectrum disorder (ASD); another ten women have children with autistic symptoms, ADD/ADHD or other developmental delays; and four women have children with other health problems, mostly immunologic. These outcomes raise concerns about the practice of postpartum vaccination and suggest that an immune mechanism may increase children's susceptibility to ASD.'
Another point is that aquired immunity from vaccination cannot be passed from mother to child through breast feeding. Only immunity from the natural disease can be passed on to a child. Now children are getting measles at an earlier age because they are no longer protected by maternal antibodies. ('Wave of Infant Measles Stems From 60's Vaccinations', Albuquerque Journal, November 1992, P.B3).
The only true immune memory comes from infection and re-challenge with the natural disease, or passively through breastfeeding.
Won't adults be at risk of catching these infections if immunity wears off?
Natural infections with measles, mumps and rubella induce lifelong immunity. Vaccination also produces very long-lasting immunity. Immune memory resulting from vaccination seems to persist even if there is no antibody detectable in the blood.
Diphtheria and tetanus are somewhat different. Protection against these diseases requires the actual presence of antibody in the blood at the time a person is exposed to the toxin. Even though immune memory lasts 40 years or more after vaccination, antibody levels decrease over time. These infectious toxins are so potent that disease can occur before the immune system has time to respond. Adults must receive boosters every 10 years to be protected against diphtheria and tetanus.
VAN UK'S Comment
MMR does not confer long term immunity!
Here is a piece in Pulse doctor's magazine
' By 2000, cases of mumps were steadily rising, increasing by 30 per cent per year compared to 1999. In some places, such as Leeds and Bradford there were increases of nine times and 30 times the number of cases between the years 2000-2001 (3). 'One third of those affected were aged over 15, just the worst time for boys to get it. In Northern Ireland 95 percent of confirmed cases were between the ages of 9 and 19.'
(Mumps outbreak shows need for MMR booster, Pulse 1 Dec 2001).
'Immunization' schedules for adults are becoming more and more complicated, because vaccine immunity doesn't last. For instance, in some countries we have DTaP for adults, MMR and MR boosters, DT boosters, polio boosters, and with the new HPV vaccine for teenage girls, they don't even know how long immunity lasts. It has been suggested that immunity lasts about 4 years - that is for a 3 dose course!
The Hep B vaccine for UK health workers (and given to US and Irish babies) is given as a booster every 5 years because it wears off!
Flu vaccines are given every year because they are considered to only work for one year.
The Advisory Committee on Immunization Practices, a division of the Centers for Disease Control and Prevention (CDC), has released the 2007-2008 recommended immunization schedules for adults in the US.
The schedule includes 11 different types of vaccines for adults, including:
* Tetanus, diphtheria, and acellular pertussis (Td/Tdap)
* Human papillomavirus (HPV)
* Measles, mumps, rubella (MMR)
* Herpes zoster (shingles)
Key changes in this year's recommendations include:
* Varicella (chickenpox) vaccination is recommended for all adults that have no apparent immunity to the virus
* Zoster (shingles) vaccination is advised for all adults 60 years of age and older, regardless of whether they have had a prior shingles episode
* HPV vaccine is recommended for women over the age of 26, who have not already completed the three-dose series
It is recommended that flu vaccination be administered to anyone with the following medical conditions:
* Chronic disorders of the cardiovascular or pulmonary systems, including asthma
* Chronic metabolic diseases, such as diabetes
* Renal or hepatic dysfunction
* Immunosuppression, including suppression caused by medications or HIV
* Pregnancy during flu season.
For Tetanus and TB, the body does not produce natural antibodies to either of these diseases. If you cannot get natural immunity, if you cannot get natural antibodies, how can you get vaccine induced ones? The immune system cannot produce antibodies to these illnesses.
Is there a risk of catching the illness from the vaccine itself?
Inactivated vaccines (such as inactivated polio vaccine or killed pertussis vaccine) and purified protein vaccines (such as diphtheria and tetanus toxoids, Hib vaccines, and hepatitis B vaccine) do not have any living organisms in them. These vaccines stimulate the immune system without causing any infection.
Live, attenuated vaccines (such as measles, mumps and rubella vaccines) infect cells and multiply in the body. However, the vaccine viruses have been sufficiently weakened, or attenuated, in the laboratory that they stimulate immunity without causing a full-blown infection. Also, they do not spread from a vaccinated child to another person.
VAN UK'S Comment
Vaccines - both inactivated and live - can cause the diseases they were meant to immunise against.
For instance, the oral polio vaccine was replaced by the injectable version because it was creating the only cases of polio in the Western world.
Here are some studies showing polio occuring after vaccination:
Albrecht RM. Poliomyelitis from a vaccinee. Lancet. 1968 Jun 22;1(7556):1371. No abstract available.PMID: 4172671; UI: 68278677.
Arya SC. Vaccine-associated poliomyelitis. Lancet. 1994 Mar 5;343(8897):610-1. No abstract available.PMID: 7906372; UI: 94150215.
Arlazoroff A. [The replacement of poliomyelitis by vaccine-associated poliomyelitis]. Harefuah. 1987 Dec 15;113(12):415-6. Hebrew. No abstract available.PMID: 3452586; UI: 88255980.
Arlazoroff A, et al. Vaccine-associated contact paralytic poliomyelitis with atypical neurological presentation. Acta Neurol Scand. 1987 Sep;76(3):210-4. PMID: 3687370; UI: 88073121.
Asindi AA, et al. Vaccine-induced polioencephalomyelitis in Scotland. Scott Med J. 1988 Aug;33(4):306-7. PMID: 2847313; UI: 89043914.
And here is one showing that DPT vaccine causes polio:
Basa, SN, "Paralytic Poliomyelitis Following Inoculation With Combined DTP Prophylactic. A review of Sixteen cases with Special Reference to Immunization Schedules in Infancy", J Indian Med Assoc, Feb 1, 1973, 60:97-99.
More recently, according to Medical writer Maria Cheng, (5 October 2007), after using oral polio vaccine in a campaign in Africa, 67 children caught paralytic polio:
A polio outbreak in Nigeria was caused by the vaccine designed to stop it, international health officials say, leaving at least 69 children paralyzed.
It is a frightening paradox in a part of the world that already distrusts western vaccines, making it even tougher to stamp out age-old diseases.
The outbreak was caused by the live polio virus that is used in vaccines given orally the preferred method in developing countries because it is cheaper and doesn't require medical training to dispense.
"This vaccine is the most effective tool we have against the virus, but it's like fighting fire with fire," said Olen Kew, a virologist at the U.S. Centers for Disease Control and Prevention.
The CDC and the World Health Organization announced the cause of the polio outbreak last week, even though they knew about it last year.
Outbreaks caused by the oral vaccine's live virus have happened before. But the continuing Nigerian outbreak is the biggest ever caused by the vaccine.
Officials now worry that the latest vaccine-caused Nigerian outbreak could trigger another vaccine scare.
Experts say such outbreaks only happen when too few children are vaccinated. In northern Nigeria, only about 39 percent of children are fully protected against polio.
The oral polio vaccine contains a weakened version of polio virus. Children who have been vaccinated excrete the virus, and in unsanitary conditions it can end up in the water supply, spreading to unvaccinated children.
(So actually the vaccinated are spreading disease to the unvaccinated, rather than the other way around).
Meningitis C vaccine - which is inactivated - has been shown to mutate and infect other people. For instance, a 16 year old Canadian girl died of meningitis B after her boyfriend was given the C vaccine and this was confirmed in a lab. Professionals are well aware that this can happen.
In a letter to the editor of the New England Journal of Medicine, January 20, 2000, German researchers had this to say. In view of the fact that an outbreak of meningococcal disease follows transmission of the meningococcus within only a few days, our report illustrates the extraordinary speed with which meningococci switch capsular serogroups. In the case we describe, the serogroup changed as a result of the transfer of serogroup-specific genes during the short period of transmission of the disease isolate. The rapidity of the serogroup switching arouses concern about the induction of herd immunity against single serogroups by vaccination programs in which capsular antigens (e.g., serogroup C polysaccharides) are used. Without lowering the incidence of meningococcal disease in the long run, such programs may rapidly increase the incidence of serogroup B meningococcal disease, for which no vaccine is available.
(i.e. the vaccine is mutating and causing meningitis B, so while meningitis C may be decreasing, meningitis B is increasing as a result) - The New England Journal of Medicine -- January 20, 2000 -- Vol. 342, No. 3 .
Another abstract from the Journal of Infectious Diseases (June, 1998) emphasized a similar concern. The appearance of serogroup B:ET15 was related temporally and geographically to mass immunization campaigns designed to control serogroup C meningococcal disease in Canada. Since there is no vaccine available to control serogroup B meningococcal disease, the appearance of this variant may have public-health significance if it demonstrates the same epidemic potential as its serogroup C counterpart.
(i.e. meningitis B was caused by the vaccine campaign for meningitis C, of which we have no vaccine).
Journal of Infectious Diseases, 1998 Jun;177(6):1754-7 (PUBMED abstract)
What about the risk of side effects from the vaccine?
The risks associated with vaccines are infinitely less than the risks associated with the diseases themselves. Fever can occur after vaccination and may cause convulsions in a few cases; but fever-induced convulsions do not cause permanent brain damage and do not increase the risk of epilepsy or any other brain disorder. The risk of convulsions is much higher after natural measles or pertussis than after vaccination.
* Pertussis Pertussis kills 1 to 3 infants every year in Canada and an equal number suffer severe brain damage. Brain damage after pertussis vaccine is extremely rare, if it occurs at all.
* Measles Measles causes encephalitis (inflammation of the brain) in about 1 out of 1,000 cases. One-third of those with measles encephalitis die and one-third survive with brain damage. Encephalitis occurs about once in every one million measles vaccinations. This occurrence rate is so low that it is unclear whether the vaccine or some other infection is responsible.
* Mumps Meningitis occurs in 1 in 10 cases of mumps. Meningitis occurs after 1 in 800,000 mumps vaccinations.
* Rubella If a woman becomes infected during the first 20 weeks of pregnancy, chances are high (8 out of 10) that the fetus will also be infected. Joint pain affects twice as many women with natural infection as women who are vaccinated.
VAN UK'S Comment
Okay, so we have 3 deaths a year from pertussis in the whole of Canada.
A 1978-79 FDA financed study showed that with DPT vaccinations, one in every 1,750 shots resulted in convulsions and shock. As five shots are required before age 5, simple arithmetic (5 divided into 1, 750) shows that there is one case of convulsions and shock for every 350 children who receive DPT vaccine. They also found that 1 in every 71,600 children died after getting DPT vaccine, there were 1 in 22,000 with acute brain disorder and 1 in 62,000 with permanent brain damage.
(Pediatrics, November 1981).
Austria, Germany and Sweden banned the pertussis vaccine since they found it caused three times as many deaths as natural pertussis (1 in 39,000 children died from the shot in West Germany and 1 in 46,000 died from the shot in Sweden). The measles vaccine was pulled from the market in Britain in 1969 (British Medical Journal, Oct. 7, 1978).
The encephalitis rate for measles is a THIRD WORLD figure, and not for Western children. Pediatrician Dr. Robert Mendelsohn said
A vaccine to prevent measles is an-other element of the MMR inoculation given in early childhood. Doctors maintain that the inoculation is necessary to prevent measles encephalitis, which they say occurs about once in 1,000 cases. After decades of experience with measles, I question this statistic, and so do many other paediatricians. The incidence of 1/1,000 may be accurate for children who live in conditions of poverty and malnutrition, but in the middle-and upper-income brackets, if one excludes simple sleepiness from the measles itself, the incidence of true encephalitis is probably more like 1/10,000 or 1/100,000.'
As stated further up in the pro-vaccinator's argument, well nourished children are more able to deal with measles than those who are not.
Vaccination can cause something called Atypical Measles Syndrome, which is more serious than wild measles and the rates of encephaltis are higher. Only vaccinated children can get this syndrome, sometimes years after the jab,
Haas EJ, et al. Atypical measles 14 years after immunization. JAMA. 1976 Aug 30;236(9):1050. No abstract available. PMID: 989583; UI: 77009302.
Scientists use vaccines to deliberately induce brain swelling in animals, so they are fully aware that they cause encephalitis at a rate much greater than 1 in a million:
Au-Jensen M, et al. Is the acute encephalopathy test in mice suited for control of pertussis vaccines? Dev Biol Stand. 1985;61:447-51. PMID: 3835081; UI: 86221312.
Animal models to control the serious neurological complications after vaccination against whooping cough are not available. In a recent paper pertussis vaccine induced acute encephalopathy in certain mouse strains (1). Healthy BALB/c mice died with shock-like symptoms after immunization with bovine serum albumin (BSA) and heat-killed pertussis. Mice not sensitized with BSA survived, and mice of strains with another H-2 type than H-2d were not susceptible. The authors concluded that the susceptibility to side effects to pertussis vaccine in mice and possibly in human is linked to the MHC. We tried to repeat the experiments reported by Steinman et al. in the hope that the murine encephalopathy model would be useful to evaluate possible neurological complications. In spite of having the same H-2d genotype, the BALB/c mice of two breeding stocks did not develop shock-like symptoms with fatal consequences after the last injection with BSA. This fact corresponds possibly with the author's observation that the pertussis vaccine encephalopathy is not under the control of H-2 genes alone. As shown in our tests the sudden deaths and encephalopathy in mice are not linked to BSA-sensitization because mice who received pertussis vaccine only showed the same symptoms as mice injected with BSA and vaccine. Histology did not indicate brain damage. It seems obvious that the deaths in our experiments were caused by the pertussis toxins present in the large numbers of bacteria given.
Munoz JJ; Peacock MG; Hadlow WJ Anaphylaxis or so-called encephalopathy in mice sensitized to an antigen.
Mumps meningitis is viral, not bacterial, and therefore not dangerous. The majority of people do not die of viral meningitis, which is considered mild.
In contrast, vaccines have actually CAUSED meningitis. For instance, the Urabe version of the MMR jab was withdrawn in 1992 because it was causing meningitis. According to American Journal of Epidemiology 2000; 151;524-530, 87 children got meningitis after being vaccinated and researchers found the risk of getting meningitis was 1 in every 14,000 doses.
The rubella schoolgirl vaccination programme was aboandoned in the UK due to being ineffective. According to Cherry (1980):
'Essentially we have controlled the disease in persons 14 or younger but we have given it a free hand in those 15 or older. Of course, the point of rubella immunisation is not prevention of rubella but preventing congenital rubella syndrome..the number of cases has declined and remained relatively stable to about 30 to 40 cases per year...this decline in CRS is curious because the number of infections in women of childbearing age has remained the same.'
(i.e. vaccine immunity wears off, leaving women of childbearing age open to getting congenital rubella syndrome. The syndrome has been reducing, but not due to vaccination because the number of infections has remained the same.
In contrast, natural immunity got as a child through having rubella, will give life-long immunity through pregnancy and protect your baby).
Other diseases like flu and chickenpox and even non-specific viruses can adversely affect a fetus, but children are not routinely vaccinated against these in the UK.
According to NHS Direct's website, 'Joint pain may develop for about a week or so, but this is more common in adults with rubella than in children.'
In contrast, the Physician's Desk Reference of 1992, states: 'Rarely have vaccine recipients developed joint problems. Following vaccination ..in women rates for arthritis are generally higher than those seen in children (12-20%) and reactions tend to be more marked and of longer duration. Symptoms may persist for a matter of months or on rare ocassions, years.'
I think up to 20% of vaccinated women getting arthritis for months or even years, is a lot more serious than getting joint pain for 'a week or so' from rubella itself.
Can vaccines cause seizures?
Yes, indirectly. Vaccines can cause fever, which can cause convulsions. Therefore, vaccine-induced fevers can cause convulsions in children who are susceptible (children whose parents or siblings have had convulsions are more likely to have a convulsion than those with no such history).
Fever from any cause triggers a convulsion in about 3% of healthy young children. Fever is the most common cause of seizures or convulsions in infants between 6 months and 6 years of age. Seizures caused by fever do not cause brain damage.
VAN UK'S Comment
That is doublespeak.
There is no such thing as 'indirectly'. A vaccine either causes seizure or it doesn't. Since the vaccine caused the fever that caused the seizure, the Vaccine caused the seizure.
This is just a play on words. Sometimes onset of seizures after vaccination start a seizure disorder, and there have been cases of brain damage after DPT vaccination:
Cherry JD, et al. Recurrent seizures after diphtheria, tetanus, and pertussis immunization. Cause and effect v temporal association. Am J Dis Child. 1984 Oct;138(10):904-7. No abstract available.PMID: 6332526; UI: 84304044.
Cherry JD et al (1993). Pertussis immunization and characteristics related to first seizures in infants and children. J Pediatr 122(6):900-3 1993. Department of Pediatrics, University of California Los Angeles School of Medicine.
In a previous study in which we examined the relationship of pertussis immunization to the onset of neurologic disorders during 1967 and 1968 and during 1972 and 1973 in Denmark, there were 554 children with initial onset of epilepsy and 2158 children with first febrile convulsions... The cause of increased severity of febrile seizures apparently associated with pertussis immunization is unknown.
Griffin MR et al (1990). Risk of seizures and encephalopathy after immunization with the diphtheria-tetanus-pertussis vaccine. JAMA 263(12):1641-5 1990. Department of Preventive Medicine, Vanderbilt University School of Medicine, Nashville, Tenn 37232-2637.
We evaluated the risks of seizures and other neurological events following diphtheria-tetanus-pertussis (DTP) immunization for 38,171 Tennessee Medicaid children who received 107,154 DTP immunizations in their first 3 years of life. There were 2 children with encephalitis; both had disease onset more than 2 weeks following DTP immunization. There were 277 children who had febrile seizures, 42 with afebrile seizures, and 37 with seizures associated with other acute neurological illness (acute symptomatic). The risk of febrile seizures in the 0 to 3 days following DTP immunization (n =6) was 1.5 (95% confidence interval, 0.6 to 3.3) times that of the control period 30 or more days following DTP immunization...'
According to the CDC, the rate for seizures after DPT is 6 out of every 10,000 doses - that's vaccines given, not per person. This works out at 1 child in 333. (http://www.cdc.gov/vaccines/pubs/vis/default.htm)
Here is a study showing that even one single seizure (that they say won't harm) negatively impacts on quality of life:
Both a single seizure and chronic recurrent seizures (epilepsy) occur commonly in childhood. Although several studies have documented the impact of pediatric epilepsy on psychosocial functioning, such as health-related quality of life (HRQOL), no studies have examined the impact of a single seizure on HRQOL. The primary objectives of this study were: (1) to compare parent–proxy HRQOL in children with a single seizure and newly diagnosed untreated epilepsy to normative data and (2) to examine differences in parent–proxy HRQOL between children with single seizure and newly diagnosed untreated epilepsy.
Results: Participants included 109 children (n = 53 single seizure; n = 56 newly diagnosed untreated epilepsy). Results indicated that both children with a single seizure and children with newly diagnosed untreated epilepsy had significant impairments in HRQOL compared to normative data. However, no significant HRQOL differences were found between the single seizure and the untreated epilepsy groups.
Discussion: Children diagnosed with a single seizure or epilepsy have similar clinically significant impairments in HRQOL. Evaluation of HRQOL, even after a first seizure, is important and will identify children at risk at the earliest opportunity, allowing for timely psychosocial intervention.
Source: Epilepsia journal, 1 June 2009.
If you think that's only for the DPT and we now have the DTaP that supposedly doesn't harm, this study says:
'The article presents comparative study of the incidence of adverse effects following vaccinations with whole cell and acellular pertussis vaccines, based on data collected in obligatory routine surveillance of AEFI in the period of 2001-2005 in Poland. Comparisons done in children less then 2-years-old show in general about twice as high incidence of adverse effects following the whole-cell than the acellular vaccine. The biggest rate of proportions (RR = 4,75) was observed for high pitch cry. There was no significant difference in incidence of the most severe reactions, including encephalopathy and nonfebrile seizures, and there was no significant difference in allergic reactions.'
(Przegl Epidemiol. 2008;62(3):589-96 - http://www.ncbi.nlm.nih.gov/pubmed/19108523).
So although it reduces some side-effects, it doesn't reduce the most serious ones.
Can vaccines cause brain damage?
Pertussis vaccine is sometimes blamed for causing brain damage in infants and young children. A review of all of the scientific evidence carried out by the Institute of Medicine in the United States found that there is no proof that pertussis vaccine causes brain damage.
Why then, has pertussis vaccine been blamed for causing brain damage? Vaccination is a very common and recognizable event in the first six months of life of most infants. Brain abnormalities, on the other hand, are uncommon and often unrecognizable in the first six months of life. Most infants who have malformations of the brain or who suffer brain damage before birth or during labour and delivery appear to be normal for the first few months of life because the brain is not fully developed. Many babies are 4 to 6 months of age or more before it becomes clear that something is wrong with their development. The diagnosis of cerebral palsy, mental retardation or developmental delay can usually not be made until the infant is several months old. By this time, the baby has already received one or more vaccinations, often with minor side effects such as fever, crying and fussiness. Since the infant appeared to be normal until the vaccine was given, the vaccine is blamed.
The following facts make it extremely unlikely that pertussis vaccine can cause brain damage:
acute illness involving the brain has not been shown to be more common after vaccination than at any other time (except for febrile or fever-induced convulsions, which do not cause brain damage);
* studies of large numbers of children have found that brain damage after vaccination is very rare, if it occurs at all;
* in searching for a link between brain damage and pertussis vaccine, no pattern of symptoms or abnormalities of laboratory tests have been found, and upon examination of the brain after death, no findings have been described that would establish pertussis vaccine as the cause of brain trauma;
* the damage in the brains of children who die of natural pertussis is caused by lack of oxygen and bleeding from small blood vessels as a result of severe coughing spells, not as a result of a toxin from the bacteria;
* no plausible mechanism has been found by which pertussis vaccination could cause brain damage.
VAN UK'S Comment
The medical profession refuse to do studies involving one group of unvaccinated vs. one group of vaccinated to see how much encephalitis and brain damage occurs in the unvaccinated.
How many parents have reported their children are brain damaged PRIOR to being given any shots? Likely none. As brain damage ALWAYS occurs AFTER vaccination, any possible association cannot be ignored.
Scientists are fully aware that vaccines cause brain damage, that's why they purposefully use vaccines to induce encephalopathy in animals:
Javier RS, et al.
Semple rabies vaccine: presence of myelin basic protein and proteolipid protein and its activity in experimental allergic encephalomyelitis. J Neurol Sci. 1989 Nov;93(2-3):221-30. PMID: 2480399; UI: 90079513.
Myelin basic protein (MBP) as a cause of postvaccinal encephalomyelitis (PVE) due to Semple rabies vaccine (SRV) has been suggested in previous reports. No actual measurement of MBP in SRV was done. In this study we detected MBP and PLP in the vaccine using immunological methods. The vaccine was found to contain 28 micrograms MBP per ml vaccine. Inoculation with SRV plus adjuvant resulted in the development of experimental allergic encephalomyelitis (EAE) in 2 of 3 guinea pigs. For control, chick embryo vaccine (CEV) was used and MBP was not detected. EAE was not induced in animals inoculated with it. These results suggest that MBP in vaccines may play a decisive role in the production of PVE.
Munoz JJ; Peacock MG; Hadlow WJ Anaphylaxis or so-called encephalopathy in mice sensitized to an antigen with the aid of pertussigen (pertussis toxin). Infect Immun, 55: 4, 1987 Apr, 1004-8
Sensitization of mice with 1 mg of bovine serum albumin (BSA) or chicken egg albumin (EA) given intraperitoneally and 300 to 400 ng of pertussigen (pertussis toxin [Ptx]) given intravenously (i.v.) induced a high degree of anaphylactic sensitivity when the mice were challenged i.v. with 1 mg of antigen 14 days later. Regardless of H-2 haplotype, all of the strains tested (CFW, BALB/cJ, DBA/2J, and C3H.SW/SnJ) were susceptible to anaphylaxis. Sensitization of mice by a multiple-dose procedure that has been reported to induce fatal encephalopathy in mice (L. Steinman, A. Weiss, N. Adelman, M. Lim, R. Zuniga, J. Oehlert, E. Hewlett, and S. Falkow, Proc. Natl. Acad. Sci. USA 82, 8733-8736, 1982) (1 mg of BSA on day -1, 100 to 400 ng of Ptx on day zero 1 mg of BSA on day +1, 100 to 400 ng of Ptx on day +2, and 1 mg of BSA on day +6) induced shock in BALB/cJ, DBA/2J, and C3H.SW/SnJ mice, but not in CFW mice. When EA was used instead of BSA, CFW, BALB/cJ, and C3H.SW/SnJ mice did not develop fatal shock, whereas DBA/2J mice did. When dose 3 of antigen (BSA or EA) was postponed to day +21, all mouse strains sensitized by the multiple-dose procedure were found to be susceptible to shock. The fatal shock induced by this procedure, as well as that induced by giving a single sensitizing dose of antigen and Ptx, could be prevented by one to three 1-ml doses of saline given i.v. at the time signs of severe shock appeared. Although only one dose of saline was often sufficient to save the mice, two or three doses were usually needed. Microscopic changes were not found in midsagittal sections of the brains of mice sensitized by either procedure. This was true of mice that died from shock or were saved from shock by injections of saline. From these results, we concluded that the proposed model for encephalopathy induced in mice by Ptx and BSA demonstrates only the well-known anaphylactogenic effect of Ptx or pertussis vaccine. Since there are many other more sensitive methods to detect Ptx, induction of anaphylaxis is not of much value for detection or quantitation of Ptx in pertussis vaccine.
Peroutka SJ; Kitamura K; Lim M; Steinman L Treatment of lethal pertussis vaccine reaction with histamine H1 antagonists. Neurology, 37: 6, 1987 Jun, 1068-72
We studied mortality after pertussis immunization in the mouse. Without treatment, 73 of 92 animals (80%) died after injection of bovine serum albumin (BSA) on day +7 of pertussis immunization. After pretreatment with 3 mg of cyproheptadine, 2 mg mianserin, or 2 mg chlorpheniramine, only 5 of 105 animals (5%) died after receiving BSA on day +7 (p less than 0.001). Blockade of histamine H1 receptors may reduce mortality in pertussis immunization-induced encephalopathy in mice.
Redhead K, et al. The activity of purified Bordetella pertussis components in murine encephalopathy. J Biol Stand. 1987 Oct;15(4):341-51. PMID: 3680302; UI: 88059141.
A murine encephalopathic syndrome can be induced by the administration of BSA and whole-cell pertussis vaccine. The present paper reports studies of the capacity of purified individual pertussis components to induce this effect. Pertussis toxin and endotoxin together with a highly immunogenic sensitizer protein were required to induce the effect. The strength of the antibody response to the sensitizer appeared to be more important than the H-2 type of the recipient in determining the susceptibility of different mouse strains. The relevance of this syndrome to the study of possible vaccine-induced encephalopathy in man is uncertain and requires further investigation.
Here are some human citations showing brain swelling after vaccination:
Barry W, Lenney W, Hatcher G, "Bulging fontanelles in infants without meningitis." Arch Dis Child 1989 Apr;64(4):635-636.
Dugmore, WN, "Bilateral Oedema at the Posterior Pole. Hypersensitivity Reaction to Alavac P injection." Br J Ophthalmol, Dec 1972, 55:848-849.
Gross TP, et al. [See Related Articles] Bulging fontanelle after immunization with diphtheria-tetanus-pertussis vaccine and diphtheria-tetanus vaccine. J Pediatr. 1989 Mar;114(3):423-5. No abstract available.PMID: 2921685; UI: 89157244.
Iwasa, S et al, "Swelling of the Brain in Mice Caused by Pertussis ... Quantitative Determination and the Responsibility of the Vaccine", Jpn J Med Sci Biol, 1985 , 38(2):53-65.
Jacob J, Mannino F, "Increased intracranial pressure after diphtheria, tetanus, and pertussis immunization." Am J Dis Child 1979 Feb;133(2):217-218.
Mathur R, Kumari S, "Bulging fontanel following triple vaccine." Indian Pediatr 1981 Jun;18(6):417-418.
Shendurnikar N, "Bulging fontanel following DPT" Indian Pediatr 1986 Nov;23(11):960.
Children who die of pertussis usually do die of lack of oxygen, rather than the bacterium, but the vaccine strain of pertussis is not the same as the wild strain and it is known to be particularly toxic. That is why the UK have now introduced DTaP vaccine (half-cell) instead of the standard DPT vaccine, as it is less potent and thought to be less likely to cause side-effects.
Can vaccination cause cancer?
Cancer is relatively uncommon in children, affecting about 1 in 10,000 children under 15 years old. But because of the marked decline in death caused by infections that used to rank No. 1 (e.g., diphtheria, pertussis, etc. ), cancer now is the second most common cause of death in children.(Accidents are No. 1.) There has been no significant increase in leukemia in children since the start of routine vaccination in the 1940s. While it is difficult to prove that immunization never causes cancer, there is no scientific evidence of a link between the two.
On the other hand, vaccination can prevent cancer, indirectly. Persons infected with hepatitis B virus are over 40 times more likely to develop cancer of the liver compared with those not infected. The vaccine prevents the infection with hepatitis B virus and this, in turn, prevents the liver cancer.
VAN UK'S Comment
Vaccines contain chemicals which are proven carcinogens.
The International Agency for Research on Cancer, (IARC) which is part of the World Health Organisation, has designated formaldehyde as a known cause of several types of throat and nasal cancer.
According to the Agency For Toxic Substances and Disease Registry, aluminium has never been evaluated to see if it causes cancer in humans. Basically they haven't done any studies to find out. You would have thought they would, considering they inject it into babies.
Some studies have implicated aluminium in causing breast cancer (as it is present in deodorants). Science Daily (September 2, 2007) stated:
A new study has identified a regionally-specific distribution of aluminium in breast tissue which may have implications for the cause of breast cancer. Scientists have found that the aluminium content of breast tissue and breast tissue fat was significantly higher in the outer regions of the breast, in close proximity to the area where there would be the highest density of antiperspirant.
Recent research has linked breast cancer with the use of aluminium-based, underarm antiperspirants. The known, but unaccounted for, higher incidence of tumours in the upper outer quadrant of the breast seemed to support such a contention. '
It was also discovered in the 1960's that the polio vaccine had been made using tissue from monkey's affected by Simian Virus 40 - a virus which was later found to caught cancer in humans. The worrying thing is, that this has passed down through the generations and SV40 cancer is being inherited by the children of those vaccinated with the affected vaccines.
Janet S. Butel (1999),1 Amy S. Arrington,1 Connie Wong,1 John A. Lednicky,1 and Milton J. Finegold2 Molecular Evidence of Simian Virus 40 Infections in Children. 1Division of Molecular Virology and 2Department of Pathology, Baylor College of Medicine, Houston, Texas The Journal of Infectious Diseases September 1999;180:884-887 © 1999 by the Infectious Diseases Society of America. All rights reserved. 0022-1899/1999/18003-0046$02.00 CONCISE COMMUNICATIONS Received 9 April 1998; revised 23 April 1999; electronically published 9 August 1999.
Recent studies have detected simian virus 40 (SV40) DNA in certain human tumors and normal tissues. The significance of human infections by SV40, which was first discovered as a contaminant of poliovirus vaccines used between 1955 and 1963, remains unknown. The occurrence of SV40 infections in unselected hospitalized children was evaluated. Polymerase chain reaction and DNA sequence analyses were done on archival tissue specimens from patients positive for SV40 neutralizing antibody. SV40 DNA was identified in samples from 4 of 20 children (1 Wilms' tumor, 3 transplanted kidney samples). Sequence variation among SV40 regulatory regions ruled out laboratory contamination of specimens. This study shows the presence of SV40 infections in pediatric patients born after 1982.
SV40 DNAs and SV40 viral isolates from monkeys and humans. J Neurovirol 1998 Apr;4(2):182-93 Division of Molecular Virology, Baylor College of Medicine, Houston, Texas 77030, USA.
SV40 DNA has been found associated with several types of human tumors. We now report a sequence comparison of SV40 DNAs from pediatric brain tumors and from osteosarcomas with viral isolates from monkeys and from humans. We analyzed the entire genomic sequences of five isolates, Baylor and VA45-54 strains from monkeys and SVCPC, SVMEN, and SVPML-1 recovered from humans, and compared them to the reference virus SV40-776. The viral sequences were highly conserved, but isolates could be distinguished by variations in the structure of the viral regulatory region and in the nucleotide sequence of the variable domain at the C-terminus of the large T-antigen gene. We conclude that multiple strains of SV40 exist that can be identified on the basis of sequences in these regions of the viral genome. The isolates were more similar to each other and to the Baylor strain than to the reference strain SV40-776. Human isolates SVCPC and SVMEN were found to be identical. The DNAs present in some human brain and bone tumors were authentic SV40 sequences. Many of the C-terminal T-ag sequences associated with human tumors were unique, but some sequences were shared by independent sources. There was no compelling evidence for human-specific strains of SV40 or for tumor type-specific associations, suggesting that SV40 has a relatively broad host range. The source of the viral DNA found in human tumors remains unknown.
Bu X, Zhang X, Zhang X, et Al. A study of simian virus 40 infection and its origin in human brain tumors. Zhonghua Liu Xing Bing Xue Za Zhi 2000 Feb;21(1):19-21. PMID 11860751.
Butel JS, Jafar S, Wong C, Arrington AS, Opekun AR, Finegold MJ, Adam E. Evidence of SV40 infections in hospitalized children. Hum Pathol 1999 Dec;30(12):1496-502. PMID 10667429.
Shivapurkar N, Harada K, Reddy J, Scheuermann RH, Xu Y, McKenna RW, Milchgrub S, Kroft SH, Feng Z, Gazdar AF. Presence of simian virus 40 DNA sequences in human lymphomas. Lancet 2002 Mar 9;359(9309):851-2. PMID 11897287.
Vilchez RA, Madden CR, Kozinetz CA, Halvorson SJ, White ZS, Jorgensen JL, Finch CJ, Butel JS. Association between simian virus 40 and non-Hodgkin lymphoma. Lancet 2002 Mar 9;359(9309):817-23. PMID 11897278.
The Leukemia and Lymphoma journal wrote in 2003:
'Millions of people worldwide were inadvertently exposed to live simian virus 40 (SV40) between 1955 and 1963 through immunization with SV40-contaminated polio vaccines. Although the prevalence of SV40 infections in humans is not known, numerous studies suggest that SV40 is a pathogen resident in the human population today. SV40 is a potent DNA tumor virus that is known to induce primary brain cancers, bone cancers, mesotheliomas, and lymphomas in laboratory animals. SV40 oncogenesis is mediated by the viral large tumor antigen (T-ag), which inactivates the tumor suppressor proteins p53 and pRb. During the last decade, independent studies using different molecular biology techniques have shown the presence of SV40 DNA, T-ag, or other viral markers in primary human brain and bone cancers and malignant mesotheliomas. Evidence suggests that there may be geographic differences in the frequency of these virus-positive tumors. Recent large independent controlled studies have shown that SV40 T-ag DNA is significantly associated with human non-Hodgkin's lymphoma (NHL). In our study, we analyzed systemic NHL from 76 HIV-1-positive and 78 HIV-1-negative patients, and nonmalignant lymphoid samples from 79 HIV-1-positive and 107 HIV-1-negative patients without tumors; 54 colon and breast carcinoma samples served as cancer controls. We used polymerase chain reaction (PCR) followed by Southern blot hybridization and DNA sequence analysis to detect DNAs of polyomaviruses and herpesviruses. SV40-specific DNA sequences were detected in 64 (42%) of 154 NHL, none of 186 nonmalignant lymphoid samples, and none of 54 control cancers. For NHL from HIV-1-positive patients, 33% contained SV40 DNA and 39% Epstein Barr virus (EBV) DNA, whereas NHLs from HIV-1-negative patients were 50% positive for SV40 and 15% positive for EBV. Few tumors were positive for both SV40 and EBV. Human herpesvirus type 8 was not detected. SV40 sequences were found most frequently in diffuse large B cell and follicular-type lymphomas. We conclude that SV40 is significantly associated with some types of NHL and that lymphomas should be added to the types of human cancers associated with SV40.'
Leuk Lymphoma. 2003;44 Suppl 3:S33-9.
Here are other medical studies showing different types of cancer brought on by other vaccines:
Allerberger, F, "An Outbreak of Suppurative Lymphadenitis Connected with BCG Vaccination in Austria- 1990/1991," Am Rev Respir Disorder, Aug 1991, 144(2) 469.
Blau HJ, et al. [Quantitative evaluation of G, A, and M immunoglobulins in children with paraplastic leukemia following BCG vaccination]. Folia Haematol Int Mag Klin Morphol Blutforsch. 1973;99(2):152-6. German. No abstract available.PMID: 4122958; UI: 73195624.
Bolognesi, DP, "Potential Leukemia Virus Subunit Vaccines: Discussion", Can Research, Feb 1976, 36(2 pt 2):655-656.
Omokoku B, Castells S, "Post-DPT inoculation cervical lymphadenitis in children." N Y State J Med 1981 Oct;81 (11):1667-1668.
Park-Dincsoy, H et al, "Lymphoid Depletion in a case of Vaccinia Gangrenosa", Laval Med, Jan 1968, 39:24-26.
Stewart, AM, et al, "Aetiology of Childhood Leukaemia", Lancet, 16 Oct, 1965, 2:789-790
Wiersbitzky S, et al. [Leukemoid reaction or leukosis after MMR preventive vaccination]. Kinderarztl Prax. 1993 May;61(3):118-9. German. No abstract available.PMID: 8326701; UI: 93316529.
Vaccination DOES NOT PREVENT cancer! Injecting yourself with toxic chemicals, heavy metals and solvants can not protect you from anything. It may be true that people who have had Hepatitis B have a greater risk of liver cancer.
1. Hepatitis B is a sexually transmitted disease, caught by people who have casual sex, drug abusers and prostitutes.
2. A baby cannot get Hepatitis B unless his mother is a carrier, which most women aren't.
3. The best way to protect yourself from liver cancer brought about by Hepatitis B is not have sex with multiple partners. Fall in love and have a commited relationship!
If you want to have casual sex, USE A CONDOM. Don't take drugs and don't engage in prostitution, and then you won't get hep B related liver cancer.
Can vaccines cause multiple sclerosis, chronic fatigue syndrome or Crohn's disease?
There are more cases of multiple sclerosis (MS) today than 30 or more years ago. The reasons for this increase are earlier and improved methods of diagnosis, improved treatment, and longer survival of MS patients. The cause of MS is not yet known. There is some evidence suggesting that infection in childhood might play a role in the development of MS. There is no evidence that immunization causes MS.
The cause of chronic fatigue syndrome is not known. Some opponents of vaccination have alleged that hepatitis B vaccination causes this illness. However studies, comparing vaccinated and unvaccinated adults failed to show any increased risk of chronic fatigue syndrome after hepatitis B vaccination.
There are many other conditions for which causes are not yet known, such as rheumatoid arthritis, Crohn's disease, ulcerative colitis and lupus erythematosus (SLE). Since most persons with these disorders were vaccinated in childhood, it is easy to blame the vaccine when no other cause can be found. However, there is no evidence to prove that these conditions are caused by vaccination.
VAN UK'S Comment
That isn't true.
The French stopped Hepatitis B vaccine for babies, after it was discovered that it was related to early onset MS.
. Mikaeloff Y, Caridade G, Rossier M et al. Hepatitis B vaccination and the risk of childhood-onset multiple sclerosis. Arch Pediatr Adolesc Med 2007; 161:1176-82.
Manslaughter charges were then brought against the vaccine companies:
3. Anon. Manslaughter charges are laid in two French drug cases. Scrip 2008; (3334):5.
Here's an interesting letter in the British Medical Journal about it (19 November 2007):
physicians have the privilege of pertaining to a profession including no less than 28% of “consultants” 1 (most of the remaining operating as “investigators”, “experts”, “opinion leaders” and all that sort of persons), thousands of hours of work on the hepatitis B vaccination inclined me (in overall accordance with some others 2) to the sad conclusion that a vast majority of our colleagues are definitely unable to pass an autonomous judgement on the quality of a study, whereas a vast majority of the remaining small minority fails to do so most probably due to lack of time: while since its public communiqué of Feb 2000, the French Agency (which is certainly not suspect of excessive criticism against this vaccination, its producers or their experts) has concluded that the results of a study by Zipp et al 3 should be no less than “rejected” due to failures some of them should be obvious at first sight, eminent scholars in that field go on quoting it as a relevant reference in the debate on the vaccine toxicity 4.
Therefore, I have tried to develop an original alternative to Evidence- Based Medicine, namely an “Idiot’s Guide to Epidemiology” in reference to the well-known series of books on computing. Let’s illustrate by an example. Fifteen years ago, just prior to the French campaign of vaccination against hepatitis B, a medical doctor like me (even practising as a “consultant”, s’il vous plaît) could have never seen a patient with multiple sclerosis (MS); today, ask any non health professional in France – a butcher, a trader, a clerk, a lawyer, a concierge, a gardener, a rep of a pharmaceutical firm: almost everybody knows one or several cases of MS in people around him/her… This does not deserve a Nobel price to guess than when a change in health environment is so dramatic, the most likely cause may be the irruption of an exogenous factor – a drug, for example: just recall the precedents of phocomelia after thalidomide introduction or pulmonary hypertension with Aminorex®.
This democratic experience of a sharp increase in the frequency of MS may be easily correlated with harder data: whereas the total number of French MS was less to 30 000 according to the last assessment available prior to the vaccination campaign 5, it was at least 60 000 in the first one delivered after the campaign 6, and a number of sources (e.g. from patients associations) give now figures close to 80 000-90 000. Interestingly enough, the French health authority – which strongly supported the vaccination campaign in 1994 – did not order any serious investigation about such a frightening human, medical as well as economical epidemics, contending itself with the vague argument that this increase would simply be an artefact due to an increase in the accuracy of diagnosis procedures… However, as due to media coverage concerning the neurological hazard of hepatitis B vaccines, the requirement to make a diagnosis of MS became stronger and stronger in France (several attacks, positive RMI, etc.), it is clear that if the real frequency had been stable, the assessed frequency should have decreased, and certainly not increased…
So, let’s try a more reasonable explanation. In its public communiqués, the French Agency always contended that if the neurological risk of vaccination “could not be excluded, it was small” – an assessment, by the way, which is notoriously devoid of any scientific meaning 7. Only in a recent publication 8, tried some of its experts to be more precise about this “smallness”, admitting that the relative risk should not be higher than a 3-fold increase – by the way an assessment strangely parallel to that by Hernan et al 9, and perfectly consistent with the assertion by the main persons in charge of the epidemiological studies performed on behalf of the French Agency who admitted that US as well as French data were consistent with an “epidemiologically important increase in risk” 10. With an recognized proportion of half of the French population exposed to hepatitis B vaccination, a background noise of less than 30 000 spontaneous MS in the overall population should have led to about 15 000 expected cases in those exposed to this vaccination (30 000 * 0.5). A 3- fold increase in risk (according to Hernan et al’s expectations 9 reluctantly confirmed by the experts of the French Agency 8 10) should thus have led to an observed number of 15 000*3 = 45 000 cases of MS in the vaccinated sub-population; added to the 15 000 expected cases in the sub-population not exposed to the vaccine, this should account (according to the “Idiot’s Guide for Arithmetic”) for a total of 60 000 cases (45 000 + 15 000) after the vaccination campaign, as exactly reflected by the most official estimates of French governmental experts 5 6, some of them directly involved in re-assuring investigations on this vaccination 11.
Due to fluctuations in current estimates, potential excess up to a total of 90 000 MS could obviously be ascribable to the additional fact that, due to vaccination of health professionals – mandatory in its principle, but unequal in its application towards lower socio-economical status (nurses, nursing auxiliaries, cleaning ladies) of high female prevalence – the “universal vaccination” has been relatively more frequent in the young female population, where the baseline incidence of MS comes to its peak, therefore accounting for an additional increase in the absolute number of cases on the basis of a 3-fold increase in risk. A small checking according to the elementary principles of arithmetic: such a frightening surge in MS prevalence is perfectly consistent with another admission by the governmental experts, namely that the number of cases spontaneously reported after hepatitis B vaccination was higher than the expected cases 12. Having regard to the known scale of underreporting in France, this admission cannot be less than the confession of an unprecedented drug-induced epidemic.
As compared to the UK, whose population is approximately the same as in France but with a higher prevalence of expected MS, and having regard to the failure of the “universal” campaign which failed to reach more than half of the French population, it can be hypothesized that a successful universal campaign in that country could, in the long term, account for a minimum of 60 000 vaccination-induced MS (to say nothing about the others hazards of this vaccination, such as lupus, myelitis, thyroid diseases, chronic fatigue, amyotrophic lateral sclerosis, etc. 13). To discuss the benefit/risk of “universal childhood immunisation”, these figures should be put in perspective with the expected number of significant complications of hepatitis B within the same country – esp. in non migrants, where the disease is generally benign and self-limited. However, I missed to find this baseline parameter in the BMA’s “call for universal childhood immunisation in the UK” 14.
As we say on this side of the Channel: “c’est trop idiot !” (that’s so silly)…
1. Campbell EG, Gruen RL, Mountford J, Miller LG, Cleary PD, Blumenthal D. A National Survey of Physician-Industry Relationships. N Engl J Med 2007; 356:1742-50.
2. Anon. Doctors and medical statistics. Lancet 2007; 370:910.
3. Zipp F, Weil JG, Einhaupl KM. No increase in demyelinating diseases after hepatitis B vaccination. Nat Med 1999; 5:964-5.
4. Hernan MA, Jick SS. Hepatitis B vaccination and multiple sclerosis: the jury is still out. Pharmacoepidemiol Drug Saf 2006; 15:653- 5.
5. Delasnerie-Laupretre N, Alperovitch A. Epidémiologie de la sclérose en plaques. Rev Prat 1991; 41:1884-7.
6. Confavreux C, Ginoux L. Scléroses en plaques. Rev Prat 2002; 52:529-37.
7. Nakao MA, Axelrod S. Numbers are better than words. Verbal specifications of frequency have no place in medicine. Am J Med 1983; 74:1061-5.
8. Hanslik T, Valleron AJ, Flahault A. [Risk-benefit assessment of hepatitis B vaccination in France, 2006]. Rev Med Interne 2006; 27:40-5.
9. Hernan M, Jick S, Olek M, Jick H. Recombinant hepatitis B vaccine and the risk of multiple sclerosis. A prospective study. Neurology 2004; 63:838-42.
10. Begaud B, Alperovitch A. Vaccinations and multiple sclerosis. N Engl J Med 2001; 344:1793.
11. Confavreux C, Suissa S, Saddier P, Bourdes V, Vukusic S. Vaccinations and the risk of relapse in multiple sclerosis. N Engl J Med 2001; 344:319-26.
12. Fourrier A, Begaud B, Alperovitch A et al. Hepatitis B vaccine and first episodes of central nervous system demyelinating disorders: a comparison between reported and expected number of cases. Br J Clin Pharmacol 2001; 51:489-90.
13. Girard M. Autoimmune hazards of hepatitis B vaccine. Autoimmun Rev 2005; 4:96-100.
14. Pollard AJ. Hepatitis B vaccination. BMJ: BMJ 2007; 335:950.
Competing interests: Dr Girard really works as an independent “consultant” for the pharmaceutical industry, including (at least until recently) hepatitis B vaccine manufacturers and a number of their competitors.
Dr. Charles Shepard from the ME Association says there is a link between ME and vaccinations:
'My research interest in this aspect of developing CFS is largely based on clinical evidence from patients seen in my practice over the past 10 years. As a result, I have gathered details on more than 200 patients with a history of either developing CFS or experiencing a significant relapse/exacerbation of CFS symptoms following a vaccination.
In addition, I have more than 150 reports referring to such a link from members of myalgic enceph-alomyelitis (ME) or CFS self-help support groups and/or their physicians throughout the world.
This data (although unpublished) suggests that tetanus, typhoid, influenza, and hepatitis B are the most commonly implicated vaccines in cases of CFS. I have reports of very few cases involving hepatitis A (using immunoglobulin), polio, or rubella vaccine, or those predominantly given during childhood—-with the possible exception of Bacillus Calmette--Guerin vaccine (three cases).
Almost all of my cases involve adults, and in a significant minority the vaccine was administered when the person had not yet fully recovered from an infective illness such as infectious mononucleosis (known as glandular fever in the U.K.) or had already experienced an adverse reaction to a previous dose of the same vaccine (as is sometimes the case with hepatitis B accine).
About one third of my cases involve vaccine-induced/exacerbated CFS following receiving the hepatitis B vaccine (HBV). Most of these patients are health care workers, particularly nurses. Most of the other patients received HBV for occupational health purposes, often as a condition of employment and without any information on side effects, such as severe neurological reactions.
The prognosis in this group has been poor, with less than 10% of the patients I have personally followed reporting any significant relief of CFS symptoms.
Although chronic debilitating fatigue is the most frequently reported symp-tom of CFS after vaccine administration in this group, around 20% also complained of significant joint pain/arthralgia, a finding consistent with several reports linking HBV to arthritis and other autoimmune disorders.5
Less than 5% of the patients also reported neurological complications/side effects such as tremors or one-sided weakness, which appear to be separate from their CFS symptoms.
For instance, one female patient developed an acute disseminated inflammation of the brain and spinal cord (encephalo-myelitis) shortly after the second dose of vaccine. This was followed by the gradual onset of CFS.
Hepatitis vaccines are highly immunogenic compounds, and a number of possible explanations exist as to why they may be more likely to trigger CFS.
One explanation involves a preexisting genetic susceptibility, which after antigenic stimulation with HBV, results in a pathological process (possibly involving immune complex formation) leading to a clinical disease.
Another explanation is that a hypersensitivity reaction occurs to a component of HBV, such as the preservative thimerosal..'
Until recently ME was unheard of. It used to be thought that people were making it up to get a day off work. It was even called 'yuppy flu'. It is obvious that something drastic is damaging people's immune systems.
Now, the Journal of Trace Elements in Medicine and Biology wrote:
'Aluminium hydroxide is used as an effective adjuvant in a wide range of vaccines for enhancing immune response to the antigen. The pathogenic role of aluminium hydroxide is now recognized by the presence of chronic fatigue syndrome, macrophagic myofasciitis and subcutaneous pseudolymphoma, linked to intramuscular injection of aluminium hydroxide-containing vaccines. The aim of this study is to verify if the subcutaneous pseudolymphoma observed in this patient in the site of vaccine injection is linked to an aluminium overload. Many years after vaccination, a subcutaneous nodule was discovered in a 45-year-old woman with subcutaneous pseudolymphoma. In skin biopsy at the injection site for vaccines, aluminium (Al) deposits are assessed by Morin stain and quantification of Al is performed by Zeeman Electrothermal Atomic Absorption Spectrophotometry. Morin stain shows Al deposits in the macrophages, and Al assays (in μg/g, dry weight) were 768.10±18 for the patient compared with the two control patients, 5.61±0.59 and 9.13±0.057. Given the pathology of this patient and the high Al concentration in skin biopsy, the authors wish to draw attention when using the Al salts known to be particularly effective as adjuvants in single or repeated vaccinations. The possible release of Al may induce other pathologies ascribed to the well-known toxicity of this metal.' - J Trace Elem Med Biol. 2012 Mar 14 - http://www.ncbi.nlm.nih.gov/pubmed/22425036).
Note the words: The pathogenic role of aluminium hydroxide is now recognized by the presence of chronic fatigue syndrome!
Here are some studies linking vaccination to Crohn's Disease:
Thompson, N.P., Montgomery, S.M.., Pounder, R.E., Wakefield, A.J., (29/4/96), Is Measles Vaccination a risk factor for inflammatory bowel disease?, The Lancet, Vol 345: 1071-1073.
Summary: Measles virus may persist in intestinal tissue, particularly that affected by Crohn’s disease, and early exposure to measles may be a risk factor for the development of Crohn’s disease. Crohn’s disease and ulcerative colitis occur in the same families and may share a common aetiology. In view of the rising incidence of inflammatory bowel disease (Crohn’s disease and ulcerative colitis), we examined the impact of measles vaccination upon these conditions. Prevalence of Crohn’s disease, ulcerative colitis, coeliac disease, and peptic ulceration were determined in 3545 people who had received live measles vaccine in 1964 as part of a measles vaccine trial. A longitudinal birth cohort of 11,407 subjects was on unvaccinated comparison cohort and 2541 partners of those vaccinated was another. Compared with the birth cohort, the relative risk of developing Crohn’s disease in the vaccinated group was 3.01 (95% CI 1.45-6.23) and of developing ulcerative colitis was 2.53 (1.15-5.58). There was not significant difference between these two groups in coeliac disease prevalence. Increased prevalence of inflammatory bowel disease, but not coeliac disease or peptic ulcerations, was found in the vaccinated cohort compared with their partners. These findings suggest that measles virus may play a part in the development not only of Crohn’s diseases by also of ulcerative colitis.
Thompson NP, Montgomery SM, Pounder RE, Wakefield AJ. Is measles vaccination a risk factor for inflammatory bowel disease?Lancet. 1995 Apr 29;345(8957):1071-4.
Here are other studies linking vaccination with bowel disease:
David A Geier, Mark R Geier MD PhD. Chronic adverse reactions associated with hepatitis B vaccination The Annals of Pharmacotherapy 2002: Vol. 36, No. 12, pp. 1970–1971.
In conclusion, our study demonstrates that adult HBV is statistically associated not only with acute neuropathy, neuritis, myelitis, vasculitis, thrombocytopenia, gastrointestinal disease, multiple sclerosis, and arthritis, but some of these patients go on to develop chronic adverse reactions that persist for at least 1 year following HBV. These types of chronic adverse reactions following adult HBV should be discussed with patients contemplating being immunized with HBV and should be included in the differential diagnosis of those who develop them following adult HBV.
Kapikian AZ. A rotavirus vaccine for prevention of severe diarrhoea of infants and young children: development, utilization and withdrawal.Novartis Found Symp. 2001;238:153-71; discussion 171-9.PMID: 11444025 [PubMed - in process]
Wiersbitzky S, Bruns R. [Toxic infant gastroenteritis acuta after DPT preventive vaccination?] Kinderarztl Prax. 1993 May;61(3):116-7. German. No abstract available. PMID: 8326700 [PubMed - indexed for MEDLINE]
Why do vaccines contain formaldehyde, aluminum, mercury, or other toxic chemicals?
Pertussis and inactivated polio vaccines are made from live bacteria or viruses that are killed with formaldehyde. Both tetanus and diphtheria toxins are inactivated with formaldehyde to make the toxoids. Following the inactivation process, purification of the vaccines removes almost all of the formaldehyde. The diphtheria, pertussis, tetanus, polio and Haemophilus b 5-in-1 vaccine contains less than 0.02% formaldehyde per dose, or less than 200 parts per million. This amount of formaldehyde is several hundred times lower than the amount known to cause harm to humans.
Some vaccines, such as the diphtheria, tetanus, pertussis and hepatitis B vaccines, are made with a mercury-containing preservative called thimerosal. It is used to prevent bacterial contamination during production of the vaccine. The amount of thimerosal is 0.01% per dose, or 100 parts per million. This compound of mercury is not toxic to humans and does not release free mercury in the body. Any vaccine that is combined with inactivated polio vaccine (IPV), such as the DPT/IPV now used in Canada, does not contain thimerosal.
Several vaccines(such as diphtheria and tetanus toxoids, and hepatitis B vaccine) contain a complex salt of aluminum called alum. The amount of aluminum is less than 1 mg per dose. This amount of aluminum is not known to cause any harm to humans. Much larger quantities of aluminum salts are taken and absorbed into the body in the form of antacids (e.g. 200-400 mg of aluminum hydroxide per tablet) without any serious side effects.
Some vaccines contain trace amounts of antibiotics used during the manufacturing process. The purpose of the antibiotics is to prevent bacterial contamination of the tissue culture cells in which the viruses are grown. Measles, mumps and rubella (MMR) vaccine and inactivated polio vaccine (IPV) each contains less than 25 ug of neomycin per dose (less than 0.000025 grams)
VAN UK'S Comment
Formaldehyde causes cancer (see above response to 'do vaccines cause cancer'?). Aluminium has been linked to breast cancer, memory loss, dementia and other conditions (see 'Vaccines and How They Are Made' page).
It is absolutely ridiculous to suggest that mercury is not toxic to babies!
According to the Environmental Protection Agency, mercury can cause:
* Impairment of the peripheral vision
* Disturbances in sensations ("pins and needles" feelings, numbness) usually in the hands feet and sometimes around the mouth
* Lack of coordination of movements, such as writing
* Impairment of speech, hearing, walking;
* Muscle weakness
* Skin rashes
* Mood swing
* Memory loss
* Mental disturbance
Health problems caused by mercury depend on how much has entered your body, how it entered your body, how long you have been exposed to it, and how your body responds to the mercury. People are at risk when they consume mercury-contaminated fish and when they are exposed to spilled mercury.
Elemental (metallic) mercury and its compounds are toxic and exposure to excessive levels can permanently damage or fatally injure the brain and kidneys. Elemental mercury can also be absorbed through the skin and cause allergic reactions. Ingestion of inorganic mercury compounds can cause severe renal and gastrointestinal toxicity. Organic compounds of mercury such as methylmercury are considered the most toxic forms of the element. Exposures to very small amounts of these compounds can result in devastating neurological damage and death.
For fetuses, infants and children, the primary health effects of mercury are on neurological development. Even low levels of mercury exposure such as result from mother's consumption methylmercury in dietary sources can adversely affect the brain and nervous system. Impacts on memory, attention, language and other skills have been found in children exposed to moderate levels in the womb.'
See http://www.epa.gov/iris/subst/0370.htm for a paper on side-effects caused by mercury exposure.
Even vaccines which are sold as thimerosal free, have trace amounts of thimerosal in them (see 'Mercury Free Vaccines Still Have Mercury In Them' page).
It is a total myth that babies can handle 'a tiny bit' of thimerosal. According to Merck vaccine manufacturer, their data sheet on thimerosal states:
'HAZARD SYMBOL T+ (VERY TOXIC). Criteria: Inhalation, swallowing or absorbtion through the skin in very small amounts can cause considerable damage to health and may sometimes be lethal.'
See here for a copy of the document:
Do vaccines contain blood, serum, animal tissue or fetal tissue?
No vaccine contains human blood or serum. Trace amounts of human albumin (a protein fractionated from whole blood) are used as a stabilizer in rabies vaccine. No vaccine contains animal or human cells. Viral vaccines are grown in cells derived from animals (chick embryo or monkey cells) or humans (fetal cells). A certain stages of production, calf serum may be added to fluid in which the cells are growing. (Calf serum is necessary for proper growth of cells in the test tube.) During purification of the vaccine, all calf serum and all cells (animal or human) are removed. Trace amounts of some proteins from the cells may remain in the vaccine.
VAN UK'S Comment
They are contradicting themselves in the same paragraph!
Human Albumin IS a human blood product, therefore vaccines contain human blood.
They say vaccines don't contain animal products, then admit they are made using them and may be in the vaccine in trace amounts. In manufacturer's data sheets, monkey kidneys and other animal ingredients are listed clearly. They do contain animal products. There is no such thing as a vegetarian vaccine.
They admit vaccines contain human fetal tissue, and this is used as a culture in exactly the same way as animal cultures are used.
Don't some vaccines contain brain tissue, which can cause mad cow diseases?
No. The only vaccine that contained brain tissue was the original rabies vaccine, which contained rabbit brain tissue. That vaccine is no longer used in Canada or the United States.
VAN UK'S Comment
This isn't true. There is bovine brain tissue in vaccines. In 1992, the Italian government banned the HIB vaccine because they were worried about possible transmission of BSE:
'The Italian ministry of health has suspended the use and marketing of a vaccine against Haemophilus influenzae type b (Hib) because of fears that it could transmit bovine spongiform encephalopathy (BSE) to humans.
The police were called in to seize batches of HibTITER from the Italian outlets of the US manufacturer Wyeth-Lederle on 17 January. The vaccine was used in the Italian national vaccination programme, but the use of bovine heart-brain infusion agar to promote bacterial growth early in the manufacturing process has worried the Commissione Unica per il Farmaco, which is part of the ministry of health.
Last year the ministry was given new powers to minimise the risk of transmission of BSE to humans after the possibility of a link between BSE and the new form of Creutzfeldt-Jakob disease was raised. It has subsequently closely monitored the manufacturing processes of all drugs and vaccines used in Italy.
Don Barret, a spokesman for Wyeth in the United Kingdom, said: "We are aghast, we do not know why the Italian government has taken this action. There is no scientific basis for it. We strongly disagree and remain thoroughly convinced of the safety of our product."
The European Agency for the Evaluation of Medicinal Products, the key advisory body overseeing safety of medicinal products for the European Union, discussed the issue on 22 January. Its Committee for Proprietary Medicinal Products (CPMP) concluded that it "remains confident about the safety of HibTITER and is reassured that the manufacturing process complies with all relevant CPMP guidelines on prevention of the risk of transmission of animal spongiform encephalopathy."
The committee said that careful examination of materials used in the manufacture of vaccines was carried out, including a review of the sources of bovine material, before the vaccine was licensed. Bovine material was used only in the first step of the manufacturing process and was not an ingredient in the finished product. The bovine material came from herds in countries free of BSE such as Australia and the US. There had been no breeding from outside the herds, and the material was obtained under veterinary supervision.
The Italian ministry of health said that there was little or no risk to the public and that people who had received the vaccine should not worry, but that under Italian law even the remotest risk of transmission of BSE was a reason to act. The ministry added that there was another Hib vaccine which was not made using bovine heart-brain infusion and that it would be used in the national vaccination programme.'
(British Medical Journal, vol.314, February 1997).
Furthermore, the Medeva oral polio vaccine was recalled in 2000 due to concern that it may transmit BSE and cause the human form, nv. CJD. According to Anthony Bevins, Political Editor of the Express newspaper, 21 October 2000,
A polIo vaccine given to seven million people was recalled on Friday because of a feared link with mad cow disease.
The Department of Health desperately played down the risk that patients - most of them children - could have been infected since 1990 with CJD, the deadly human version of BSE.
Chief Medical Officer Professor Liam Donaldson said: "I am advised that the risk of a person contracting the disease from this oral polio vaccine is incalculably small." He said he and his family had all had the vaccine and added: "Parents of children who've had vaccines in the past can be reassured that there is no major safety issue."
But with the BSE inquiry report due to be published on Thursday, distrust of official reassurances is high. Concern over the risks of contracting CJD from food has risen 17 points to 43 per cent in the past year, according to a survey out this week.
Public worries will be increased by the fact that Parliament has repeatedly been misinformed about the oral polio vaccine banned yesterday because it was based on UK-sourced bovine material. Such material was said to have been eradicated from all vaccines in 1989.
Only last March, Health Minister Yvette Cooper told Liberal Democrat MP Norman Baker in the Commons: "I am advised that all vaccine manufacturers were contacted in 1989 and that they decided immediately to source their bovine materials outside the UK."
The risk isn't just in the UK, either. According to the Ottawa Citizen, March 1999, soldiers were put at risk of developing CJD after having Hepatitis A vaccinations:
Maj. Dan Drew's seven months in the former Yugoslavia were hell on Earth. He survived the worst battle Canadian soldiers had fought since the Korean War when his battalion took part in a firefight that pitted his fellow soldiers against a Croat army.
But now, six years later, Maj. Drew wonders whether the most serious threat to his life occurred before he even set foot on Balkan soil.
He worries that his blood and the blood of thousands of other Canadian soldiers was accidentally contaminated with a fatal brain malady called Creutzfeldt-Jakob disease. And there is no way he will ever know for sure because no detailed military records exist to show which soldiers received the contaminated batch of vaccine.
"We've been exposed to a potential death sentence," Maj. Drew says.
In January 1993, Maj. Drew was one of thousands of soldiers inoculated against Hepatitis A with a batch of gamma globulin immune serum that may have been tainted with CJD, the human form of the so-called "mad cow" disease, the Citizen has learned. '
Won't breastfeeding and good nutrition prevent these childhood diseases?
Breastfeeding is not an alternative to infant vaccination, and it does not enhance the responses to vaccination. Breastfeeding provides some protection against many infections because special antibodies are made in the breast and are present in human breast milk. Babies who are breastfed generally have lower rates of many infections including viral respiratory infections, ear infections and diarrhea. The protection provided by breast milk is incomplete and can be overcome if the baby is exposed to a large amount of a disease-causing organism. Moreover, the protection disappears rapidly as soon as breastfeeding stops.
Good nutrition helps the body's defences against infection to function normally. Infections are more severe in anyone with poor nutrition. Special immune cells called lymphocytes are easily damaged if one's diet does not include enough protein. For this reason, malnourished children are much more likely to die of infections such as measles or pertussis than well-nourished children. Vitamin A deficiency, in particular, greatly increases the risk of severe illness.
VAN UK'S Comment
How DARE they say breast milk is incomplete!!
Your breast milk provides EVERYTHING your baby needs for survival. Nature did not make a mistake. Studies have shown that breast milk can protect against all the diseases currently vaccinated against.
Breast milk immunity does not stop when you stop breast feeding. Studies have also shown them to last for years after you cease feeding.
Children who have been breast fed are also less likely to get allergies, cancers, diabetes, heart disease and weight problems as adults, so the benefits are for life.
Doctors and drug companies will try to make you feel as if you need them for the survival of your baby. You don't. You are all he needs.
'If a mother breast feeds whilst she is ill, she will have antibodies to her illness in her breast milk, and these will pass onto her baby and protect him from contracting the illness.
This is especially important in the first few months of life when the babys antibody and immunity system is still forming.
At the time of babys first vaccinations at 2 months, the baby will still not have developed a mature immune system or even have enough quantities of his own antibodies to cope with it.
This is thought to be a reason why some babies react badly to vaccinations and why over 70% of SIDS cases occur in bottle fed, vaccinated babies. These babies will not have had the added benefit of mums antibodies to help them fight vaccine toxins.
The best protection your baby has against illnesses is from you! The IgA (antibody) you produce against infectious disease is made during the latter part of your pregnancy, during the birth and through the whole time you are breast feeding him.
This will protect him against polio. Rather ironically, the first study done into the anti-polio properties of breast milk was by Albert Sabin, inventor of the live oral polio vaccine.
He infected mice with polio virus type 2 and then took breast milk from 71 American women and fed it to the mice. The milk had an 84% success rate at neutralising polio virus.
It also neutralised many other viruses such as yellow fever, dengue, japanese encephalitis and west nile virus regardless of whether the mother had been exposed to any of these illnesses or not. (3).
About 80 percent of the cells in breast milk are macrophages, cells that kill bacteria, fungi and viruses. Breast-fed babies are protected, in varying degrees, from a number of illnesses, including pneumonia, botulism, bronchitis, staphylococcal infections, influenza, ear infections, and German measles. Furthermore, mothers produce antibodies to whatever disease is present in their environment, making their milk custom-designed to fight the diseases their babies are exposed to as well. (14).
According to Dr. Penny Stanway:
Breast feeding could save the lives of hundreds of thousands of children who die around the world because they are bottle-fed.
Breast fed babies are less likely to die before their third birthday because they are less likely to get life-threatening illnesses and if they do get ill, they are better able to cope with it. (21).
Breast feeding protects against gut disorders, chest infections and urinary infections.
Infective diarrhoea, pneumonia, meningitis and septicaemia are also more common in bottle fed infants.
In other infections, such as Haemophilius Influenzae B, breast feeding for longer than 6 months has been found to have a protective effect (Takala et al, 1989). (15).
A study showed that a bottle fed baby is more likely to be admitted to hospital - regardless of its parents social status. Bottle fed babies of less educated mothers were also four times more likely to catch infection than their breast fed peers.
Hib are RSV are also more common in formula fed babies. Haemophilius organisms stick to cells on the breathing passages.
Breast milk contains sugars known as oligosaccharides. These are absorbed into the body and they line the lungs where they prevent harmful organisms from doing damage. Formula fed babies are denied this protection. (4).
Here are some more studies which have shown how your milk protects your baby from disease:
Tropical Paediatrics, 1989, reported that breast milk samples contained significant amounts of antibodies to pertussis (whooping cough), Hib, strep B and meningitis.
The IgA antibody levels were higher than in both maternal and infant serums
samples may indicate a protective role for breast milk against the four infections of early childhood.
That means that not only does your breast milk protect IT IS BETTER AND MORE EFFECTIVE THAN A VACCINE. (5).
Journal of Epidemiology, 1997, reported that breast milk protects your baby against Hib for up to 10 years after lactation has ceased.
For each week of breast feeding, the protection improved. (6).
According to Pediatrics, 2005:
Research in developed and developing countries of the world, including middle-class populations in developed countries, provides strong evidence that human milk feeding decreases the incidence and/or severity of a wide range of infectious diseases including bacterial meningitis, bacteremia,diarrhea, respiratory tract infection, necrotizing enterocolitis, otitis media, urinary tract infection, and late-onset sepsis in preterm infants. In addition, postneonatal infant mortality rates in the United States are reduced by 21% in breastfed infants.
Some studies suggest decreased rates of sudden infant death syndrome in the first year of life and reduction in incidence of insulin-dependent (type 1) and noninsulin-dependent (type 2) diabetes mellitus, lymphoma, leukaemia, and Hodgkin disease, overweight and obesity, hypercholesterolemia, and asthma in older children and adults who were breastfed, compared with individuals who were not breastfed. Additional research in this area is warranted.'
(Taken from my book, 'Breast Milk: A Natural Immunisation', see Vaccine Books page for details).
Money raised from the book goes to upkeep this website and to buy more books to sell to the public. I don't keep proceeds for myself, except if there are travel expenses to go and run stalls and things, which I only do a couple of times a year.
I have no competing interests. I just love babies.
Aren't the only children who die of these infections suffering from malnutrition or defects of the immune system?
Although infections such as measles and pertussis are much more likely to kill a child who is malnourished, these infections can also kill healthy, well-nourished children.
VAN UK'S COMMENT:
In the majority of cases, complications and deaths from childhood illnesses occur in those with pre-existing illnesses or disabilities, and those already vaccinated:
'According to the DOH, in their book Immunisation Against Infectious Diseases,Before 1988 (when the MMR was introduced) more than half the acute measles deaths occurred in previously healthy children who had not been immunised. They quote the study C L MILLER. Deaths from measles in England and Wales, 1970-83. British Medical Journal, Vol 290, 9 February 1985, but if you actually read this study (which they are relying on parents not doing), you will find it actually says:
No attempt was made to establish further clinical details, vaccination history, or social class. - i.e. they didnt know the vaccine status of the individuals. And: 90% of deaths in those previously normal occurred in those over the age of 15 months, when the vaccines are usually given. These children were probably vaccinated prior to dying of measles as they were of vaccination age.
Nearly half the children who died were grossly physically or mentally abnormal or both. The pre-existing conditions in the 126 previously abnormal individuals included cerebral palsy (24), mental retardation (20), Down's syndrome (19) and various congenital abnormalities (22). There were nine children with immune deficiency or immunosuppression, and 19 aged 2-8 with lymphatic leukaemia, a number of them in remission.'
It is true that very rarely, a healthy child may die of a childhood illness. But to put this in context, there are far more children who die in car accidents than ever died of a childhood disease, yet we don't stop driving our children around in cars.
The leading cause of childhood death in USA between 2002-2004 was:
Accidents - 4.6% per 100,000
Cancer - 3.2% per 100,000
Congenital Abnormalties - 1.1% per 100,000
Murder - 0.8% per 100,000
Suicide - 0.4% per 100,000
Your child has a far higher chance of dying of cancer than a childhood disease. Yet in the 19th and early 20th centuries, it was virtually unheard of. It may be that through vaccinating, we are swapping our infectious disease death rates for cancer death rates.
Don't infections like measles stimulate the immune system and lead to better overall health?
No. Natural infection with measles does not provide a general form of stimulation of the immune system. It stimulates immunity to measles only. No infection acts as a general stimulus to the immune system. There is no scientific evidence that infection with measles or any other germ is necessary or important for natural and healthy development of the immune system.
Furthermore, what we know of measles makes it extremely unlikely that measles plays any role whatsoever in the normal development of the human immune system. Measles infection results in marked suppression of many parts of the immune system, which lasts for several months. During this time, the child is more susceptible to a number of other infections. This suppression of the immune system caused by measles actually leads to the high rate of other infections that complicate measles.
VAN UK'S Comment
That isn't true. There have been studies which show that having childhood illnesses can stimulate the immune system.
Children who have measles are less likely to develop asthma later in life. According to research from the University of Aberdeen, scientists have found that early exposure to measles appeared to protect against the allergic condition.
The team studied 300 people from 1964, and monitored their allergic responses and immune systems regularly, over 30 years.
Suspicions are growing amongst the medical profession that the growing incidence of asthma in Britain is connected to the cosseting of children from disease. Some research has suggested that exposure to infections such as measles, hepatitis A and TB may prevent allergic conditions.
(reported in the Daily Telegraph, 19 April 2000).
Here are some other studies showing measles strengthening the immune system:
A 6-year-old girl suffering from severe psoriasis had been treated unsuccessfully by various conventional methods. She developed measles and, on recovery from measles, the psoriasis soon cleared up and now, 6 months later, she still has had no further recurrence. The basic defect in psoriasis, basal cell hyperplasia and defective keratinization, may well be immunologically mediated. Measles virus, by its immunosuppressive effect can lead to remission of psoriasis.
Chakravarti VS, Lingam S. Measles induced remission of psoriasis. Ann Trop Paediatr. 1986 Dec;6(4):293-4.
Kids Who Have Measles Have Less Allergies:
OBJECTIVE: Viral and bacterial infections in childhood decrease the likelihood of allergic diseases in later life. The frequency of allergic diseases in patients with a history of measles has been reported to be low but some studies still suggest that measles can increase the frequency of allergic diseases. The aim of this study was to investigate the frequency of allergic diseases following measles in childhood. METHODS: Fifty-two children hospitalized in our clinic with measles were compared with 51 children without measles. Allergic diseases were investigated in both groups by using the International Study of Asthma and Allergies in Childhood (ISAAC) questionnaire. In all children, allergy skin tests were performed with the four most common allergens. RESULTS: Sensitivity to Dermatophagoides pteronyssinus was less frequent in children with measles than in those without (p < 0.05). A history of nebulized salbutamol use in the emergency room in the previous 12 months was also less frequent in the measles group (p < 0.05). Inhaled corticosteroid use was more common in the group without measles (p < 0.05). CONCLUSION: The results of this study indicate that findings of allergic disease are less frequent in children with a history of measles. These children were less sensitive to D. pteronyssinus.
Allergol Immunopathol (Madr). 2006 Jul-Aug;34(4):146-9.
BACKGROUND: Epidemiological studies have led to speculation that infections in early childhood may prevent allergic sensitisation but evidence to support this hypothesis is lacking. We investigated whether measles infection protects against the development of atopy in children of Guinea-Bissau, West Africa. METHODS: We conducted a historical cohort study in Bandim, a semi-rural district of Bissau, the capital of Guinea-Bissau. 395 young adults, first surveyed in 1978-80 aged 0-6 years, were followed up in 1994. Our analyses were restricted to 262 individuals still living in Bandim for whom a measles history, documented in childhood, was judged to be reliable. We defined atopy as skin-prick test positivity (> or = 3 mm weal) to one or more of seven allergens. FINDINGS: 17 (12.8 percent) of 133 participants who had had measles infection were atopic compared with 33 (25.6 percent) of 129 of those who had been vaccinated and not had measles (odds ratio, adjusted for potential confounding variables 0.36 [95 percent CI 0.17-0.78], p=O.O1). Participants who had been breastfed for more than a year were less likely to have a positive skin test to housedust mite. After adjustment for breastfeeding and other variables, measles infection was associated with a large reduction in the risk of skin-prick test positivity to housedust mite (odds ratio for Dermatophagoides pteronyssinus 0.20 [0.05-0.81], p=0.02; D farinae 0.20 [0.06-0.71], p=0.01). INTERPRETATION: Measles infection may prevent the development of atopy in African children.
Measles and atopy in Guinea-Bissau. Lancet. 1996 Jun 29;347(9018):1792-6.
Seizure Disorders DISAPPEARED after infection with rotavirus, measles and mumps:
In general, epileptic seizures become more serious following infections. However, transient and permanent improvement of epileptic seizures has been observed following acute viral infections, without a recent change in anti-epileptic therapy. Questionnaires were sent to 73 institutions, throughout Japan, where pediatric neurologists care for children with epilepsy to characterize this phenomenon through clinician survey. Completed surveys were received from 11 institutions, and 21 cases were selected for the study. The age of the patients were 6 months to 17 years. The West syndrome or epilepsy subsequent to West syndrome cases were 16 out of 21. Two cases of symptomatic generalized epilepsy and one case each of symptomatic partial epilepsy, continuous spike-waves of slow sleep and severe myoclonic epilepsy in infancy were also reported. These seizures disappeared within 2 weeks subsequent to viral infections such as, exanthema subitum, rotavirus colitis, measles and mumps. The disappearance of intractable epileptic seizures following acute viral infections might be related to the inflammatory processes or the increased levels of antibodies after viral infections.
Hitoshi Yamamoto, et al. Spontaneous improvement of intractable epileptic seizures following acute viral infections. Brain and Development
Volume 26, Issue 6, September 2004, Pages 377-379.
Positive Effects of Natural Rotavirus Infection
Epidemiologic studies have demonstrated that children who acquire natural rotavirus infections develop immunity to subsequent infections, with the protective effect increasing with each natural infection. Natural infections also decrease the severity of any subsequent rotavirus infections. Notably, asymptomatic infections provide protection similar to that induced by symptomatic infections. Data also suggest that the antibody response to natural infection is heterotypic, and therefore may provide protection against multiple serotypes.
(The Pediatric Infectious Disease Journal:Volume 28(3) SupplementMarch 2009pp S54-S56).
Is vaccination safer when my child is older, rather than at 2 months of age?
There is no evidence that side effects from vaccination are more common in younger infants. The purpose of starting vaccination at 2 months of age is to protect the child against pertussis and Haemophilus b disease as early in life as possible. Complications and deaths from pertussis are most common in infants less than 6 months of age. Infants can respond to vaccination at a very young age.
VAN UK'S Comment
That's not true.
In 1975 and 1976, the Japanese increased the vaccine starting age to 2 years old, and cot death disappeared. They jumped from 17th place in infant death rate, to the lowest rate in the world.
In 1988 they lowered the vaccination age to 3 months old, and cot death rates rose again.
According to One World South Asia, child death rates have lowered at the same time as 'immunisation' rates:
May 11, 2006
Two recent health surveys carried out by the Government have thrown up mixed results. While one reports that the Infant Mortality Rate has fallen below 60 for the first time in the country, the worrying sign is that the already low immunisation rates are showing further decline.
The most alarming is the case of Uttar Pradesh, which shows a fall in immunisation from 43.7 per cent in 1998-99 to 28.1 per cent in the latest data.
In 1998-1999, 54 per cent of the children in the country were reported to be fully immunised. But a district household survey 2002-2004, the data for which was released last month, shows a decline in this to 47.6 per cent. In 1989-99, India had one-third of the world?s non-immunised children.
Immunisation rates seem to have fallen across the country, including Uttar Pradesh and Bihar, which account for 40 per cent of the total children in the age group of zero to one who need immunisation. But unlike Uttar Pradesh, Bihar has shown only a marginal decline, from 24.4 to 22.4 per cent.
Experts believe that the focus on polio eradication, at the cost of routine immunisation, could have contributed to the decline.
The other states showing low figures are Rajasthan (25.4 per cent), Tripura (26.7 per cent), Jharkhand (29.3 per cent) and Madhya Pradesh (32.5 per cent).
The states at the other end of the spectrum are Tamil Nadu (with an immunisation rate of 92.1 per cent), Kerala (81.2 per cent), Pondicherry (89.4 per cent), Goa (81.5 per cent) and Himachal Pradesh (79.4 per cent).
There is good news, however, on the infant mortality front. For the first time, India has reported IMR below 60, with the survey from Registrar General of India released recently showing 58 deaths per 1,000 live births in the country.
Though the rates are still high compared to other countries, the figures have shown decline from 68/1,000 live births in 2000, and 60/1,000 live births in 2004.' (Indian Express).
The infant's immune system is not fully developed until he is 6 years old, and when he is newborn, he doesn't have his own immune system. He relies on antibodies left over from the placenta, and his mother's breast milk. Very young babies who are vaccinated can have complications and even death.
According to Dr. Torch, in a study of 103 cot deaths, 6.5% occured within 12 hours of vaccination, 13% within 24 hours and a staggering 70% within 3 weeks of DPT vaccination.
(Torch, W.S., 1982. Diphtheria-pertussis-tetanus (DPT) immunization: a potential cause of the Sudden Infant Death Syndrome (SIDS). Neurology; 32(4): A169 abstract).
Here's some more studies showing young babies dying after vaccination:
Baraff LJ, et al (1983) Possible temporal association between diphtheria-tetanus toxoid-pertussis vaccination and sudden infant death syndrome. Pediatr Infect Dis. 1983 Jan-Feb;2(1):7-11. PMID: 6835859; UI: 83169234.
Because diphtheria and tetanus toxoids pertussis (DTP) vaccine is routinely given during the period of highest incidence of sudden infant death syndrome (SIDS), this study was undertaken to determine if there is a temporal association between DTP immunization and SIDS. Parents of 145 SIDS victims who died in Los Angeles County between January 1, 1979, and August 23, 1980, were contacted and interviewed regarding their child's recent immunization history. Fifty-three had received a DTP immunization. Of these 53, 27 had received a DTP immunization within 28 days of death. Six SIDS deaths occurred within 24 hours and 17 occurred within 1 week of DTP immunization. These SIDS deaths were significantly more than expected were there no association between DTP immunization and SIDS. An additional 46 infants had a physician/clinic visit without DTP immunization prior to death. Forty of these infants died within 28 days of this visit, seven on the third day and 22 within the first week following the visit. These deaths were also significantly more than expected. These data suggest a temporal association between DTP immunization, physician visits without DTP immunization and SIDS. PMID: 6835859, UI: 83169234 "They found a statistically significant excess of deaths in the first day and the first week after vaccination, i.e., a "temporal association." They rejected the use of a "control group," and instead relied on the intuitively obvious assumption that "there should be no temporal association between DPT immunization and SIDS were there no causal relationship between these two events." I have not found any criticism of this article for relying on "anecdotal evidence." This study was not financed by the US Government but apparently by the UCLA School of Medicine and the Los Angeles County Department of Health Services."--Harris Coulter
Geraghty KC. DTP immunization and SIDS.J Pediatr. 1984 Jul;105(1):169-71. No abstract available.PMID: 6610735 [PubMed - indexed for MEDLINE]
Torch, W.C., 1986 a. Characteristics of diphtheria-pertussis-tetanus (DPT) postvaccinal deaths and DPT-caused Sudden Infant Deaths Syndrome (SIDS): a review. Neurology (suppl 1); 36: 148 (abstract).
Torch, W.C., 1986 b. Diphtheria-pertussis-tetanus (DPT) imunization may be an unrecognized cause of Sudden Infant Death (SIDS) and Near-Miss Syndrome (NMS): 12 case reports. Neurology (suppl 1); 36: 149 (abstract).
The peak rates for cot death occur between the ages of 2 and 6 months, when the vaccines are being given.
CDC Says They CAN'T Counter Anti-Vaccine Arguments!
Interesting snip bits from CDC document:
'The number of reported adverse events associated with vaccination was greater than the number of reported cases of vaccine-preventible disease.'
'CDC does not have complete adverse event surveillance data on which to base health messages.'
I.e. we don't really know that vaccines are safe when we tell you that as we don't properly monitor side-effects.
'Some claims against vaccine CANNOT be disproved.'
Of course the document is not about how to improve vaccine safety but merely how to counter perfectly valid arguments of 'anti vaccine' parents who are obviously all into alternative therapies and 'hostile' towards doctors.
Well, CDC if you read this, I have news for you. I was not 'into' alternative therapies when myself and my husband decided not to vaccinate. I was 18 and had never encountered alternative therapies at that time.
I was not hostile towards my doctor. They were hostile towards me for opting out of a programme that is voluntary anyway. They called me names, they yelled at me, they threatened me, they refused to see my children even for genuine medical problems unless I agreed to vaccinate, they denied me ante-natal care until I was 6 months pregnant with my second child.
I have lovely allopathic doctors now, that I use in addition to alternative therapy when the need arises and if you actually talked to parents who don't vaccinate, you would find similar stories.
Source: See http://www.rescuepost.com/files/jim-moody-post-swot-safety.pdf for CDC Document.